- The occupation of receptors by an antagonist so that usual agonists are relatively ineffective.
- Receptor blockade, blocking the effect of a hormone at the cell surface.
- Arrest of nerve impulse conduction or transmission at autonomic synaptic junctions, autonomic receptor sites, or neuromuscular junctions by various means, most often pharmacotherapy.
- Intravenous injection of large amounts of colloidal dyes or other substances to block reticuloendothelial cells (phagocytosis is temporarily prevented).
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Examples: glitazone, GI cocktail, etc.