Luxiq Side Effects

Please note - some side effects for Luxiq may not be reported. Always consult your doctor or healthcare specialist for medical advice. You may also report side effects to the FDA at http://www.fda.gov/medwatch/ or 1-800-FDA-1088 (1-800-332-1088).

Side Effects of Luxiq - for the Consumer

Luxiq Foam

All medicines may cause side effects, but many people have no, or minor, side effects. Check with your doctor if any of these most COMMON side effects persist or become bothersome when using Luxiq Foam:

Dry skin; mild, temporary stinging when applied.

Severe allergic reactions (rash; hives; itching; difficulty breathing; tightness in the chest; swelling of the mouth, face, lips, or tongue); acne-like rash; burning, cracking, irritation, or peeling not present before you began using Luxiq Foam; excessive hair growth; inflamed hair follicles; inflammation around the mouth; muscle weakness; thinning, softening, or discoloration of the skin; unusual weight gain, especially in the face.

Luxiq Side Effects - for the Professional

Luxiq

The most frequent adverse event was burning/itching/stinging at the application site; the incidence and severity of this event were as follows:

Other adverse events which were considered to be possibly, probably, or definitely related to Luxíq occurred in 1 patient each; these were paresthesia, pruritus, acne, alopecia, and conjunctivitis.

The following additional local adverse reactions have been reported with topical corticosteroids, and they may occur more frequently with the use of occlusive dressings. These reactions are listed in an approximately decreasing order of occurrence: irritation; dryness; folliculitis; acneiform eruptions; hypopigmentation; perioral dermatitis; allergic contact dermatitis; secondary infection; skin atrophy; striae; and miliaria.

Systemic absorption of topical corticosteroids has produced reversible hypothalamic-pituitary-adrenal (HPA) axis suppression, manifestations of Cushing’s syndrome, hyperglycemia, and glucosuria in some patients.

Side Effects by Body System

Local

Skin atrophy may become evident within one to two months of use and is due to the inhibitory effect of corticosteroids on collagen formation. Skin on the face, axilla, and groin appear to be most susceptible to the adverse long-term effects of topical betamethasone. Use of high potency topical corticosteroids on these areas should be minimized or avoided.

Topical corticosteroid use may impair local immune response rendering the skin more susceptible to infections. Folliculitis has occasionally been reported.

Perioral dermatitis or rosacea-like dermatitis has occurred in patients treated with potent topical corticosteroids. These conditions may flare temporarily upon discontinuation of topical steroids, prompting patients to continue their use. If topical corticosteroids are discontinued, this flare and the initial dermatitis generally resolve over a few weeks.

Worsening of psoriasis has occurred in a few patients.

Local side effects have included burning, itching, dryness, and irritation, especially when applied to denuded skin. Long-term use of topical corticosteroids may result in skin atrophy and thinning, and the development of striae, telangiectasia, subcutaneous hemorrhage, and easy bruising and bleeding. Allergic contact dermatitis has occasionally been reported.

Endocrine

Adrenal suppression has been reported in patients with psoriasis using augmented betamethasone dipropionate at doses of approximately 50 grams per week. Plasma cortisol concentrations generally return to normal within one to two weeks following discontinuation of the drug, and in some cases return to normal during continued therapy. In a few cases adrenal failure has persisted up to four months.

If betamethasone dipropionate is to be used for an extended period of time, adrenal function should be evaluated periodically. Supplemental systemic steroids may be necessary during times of stress. Other less potent forms of betamethasone may cause adrenal suppression if used on large areas or with occlusive dressings.

Endocrine side effects have included suppression of the hypothalamic-pituitary-adrenal (HPA) axis, resulting in cushingoid features and symptoms of adrenal suppression following withdrawal of the drug. These effects are more likely when higher potency topical corticosteroids are used over extensive areas or when occlusive dressing are used. In addition, augmented betamethasone, and the ointment and emollient cream formulation of betamethasone generally provide better penetration, and thus, higher risk of adrenal suppression.

Ocular

Steroid-induced cataracts and glaucoma have been reported, most often in patients receiving betamethasone eyedrops (not available in the US). In one patient, permanent eye damage resulted from long term application of betamethasone cream to the eyelids.

Ocular side effects have included rare reports of glaucoma in patients using betamethasone on the face for long periods of time. Intraocular pressure does not always return to normal following discontinuation of the drug.

Musculoskeletal

Musculoskeletal side effects have included rare reports of osteoporosis. Avascular necrosis of the hips and vertebral fractures have been documented.

Dermatologic

Dermatologic side effects have included erythema, folliculitis, pruritus, acneiform eruptions, hypopigmentation, perioral dermatitis, allergic contact dermatitis, secondary infection, miliaria, hypertrichosis, and vesiculation.

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